Physician
Pathology
Dr. Steven Yea, MD
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Steven practices Pathology. Steven is a published MD. He published 17 publications, including: 'Transforming growth factor-beta 1 (TGF-beta1) promotes IL-2 mRNA expression through the up-regulation of NF-kappaB, AP-1 and NF-AT in EL4 cells.' Steven is registered with Medicare.
Publications
- Role of nuclear factor of activated T-cells and activator protein-1 in the inhibition of interleukin-2 gene transcription by cannabinol in EL4 T-cells.
- The -238 tumor necrosis factor-alpha promoter polymorphism is associated with decreased susceptibility to cancers.
- Suppression of the interleukin-2 gene expression by aflatoxin B1 is mediated through the down-regulation of the NF-AT and AP-1 transcription factors.
- Suppression of IL-2 and IL-4 gene expression by nodularin through the reduced NF-AT binding activity.
- Microcystin-induced down-regulation of lymphocyte functions through reduced IL-2 mRNA stability.
- Inhibition of NF-kappa B/Rel nuclear translocation by dexamethasone: mechanism for the inhibition of iNOS gene expression.
- Transforming growth factor-beta 1 (TGF-beta1) promotes IL-2 mRNA expression through the up-regulation of NF-kappaB, AP-1 and NF-AT in EL4 cells.
- Tumor suppressor activity of KLF6 mediated by downregulation of the PTTG1 oncogene.
- A DDX5 S480A polymorphism is associated with increased transcription of fibrogenic genes in hepatic stellate cells.
- Ras pathway activation in hepatocellular carcinoma and anti-tumoral effect of combined sorafenib and rapamycin in vivo.
- Functional linkage of cirrhosis-predictive single nucleotide polymorphisms of Toll-like receptor 4 to hepatic stellate cell responses.
- Pivotal role of mTOR signaling in hepatocellular carcinoma.
- Ras promotes growth by alternative splicing-mediated inactivation of the KLF6 tumor suppressor in hepatocellular carcinoma.
- Downregulation of KLF6 is an early event in hepatocarcinogenesis, and stimulates proliferation while reducing differentiation.
- Regulation of Kruppel-like factor 6 tumor suppressor activity by acetylation.
- Krüppel-like factor-6 promotes preadipocyte differentiation through histone deacetylase 3-dependent repression of DLK1.
Schools
Mount Sinai School of Medicine
Doctors Specialties
- Hematology
- Hematopathology
- Pathology
Accepted Insurances
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1275761777 |
| Last Name Of The Provider | YEA |
| First Name Of The Provider | STEVEN |
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