Associate Professor, Anesthesiology, University of Colorado School of Medicine

Anesthesiology

Tobias Eckle works in the field of Anesthesiology. He has 1 awards "CMS Meaningful Use Stage 1 Certification". Tobias Eckle, MD is also published. He has 56 publications published. The lastest publication: "The impact of sedation protocols on outcomes in critical illness.' Tobias Eckle is registered with Medicare and accepts Medicare payments.

Publications

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• The impact of sedation protocols on outcomes in critical illness.
• Next Generation of Cardiovascular Studies: Transcriptional Responses of the Human Myocardium during Cardiac Surgery.
• Health Implications of Disrupted Circadian Rhythms and the Potential for Daylight as Therapy.
• Circadian Rhythms in Anesthesia and Critical Care Medicine: Potential Importance of Circadian Disruptions.
• About Dogs, Mice, and Men: From Ischemic Preconditioning to Anesthetic Postconditioning of the Heart.
• Anesthetic Cardioprotection: The Role of Adenosine.
• Identification of Hypoxia-Inducible Factor HIF-1A as Transcriptional Regulator of the A2B Adenosine Receptor during Acute Lung Injury.
• HIF1A Reduces Acute Lung Injury by Optimizing Carbohydrate Metabolism in the Alveolar Epithelium.
• Crosstalk between the equilibrative nucleoside transporter ENT2 and alveolar Adora2b adenosine receptors dampens acute lung injury.
• Attenuating myocardial ischemia by targeting A2B adenosine receptors.
• Cardiac Period 2 in myocardial ischemia: Clinical implications of a light dependent protein.
• Transesophageal echocardiography in the diagnosis of acute pericardial tamponade during hiatal hernia repair.
• Myocardial ischemia reperfusion injury: from basic science to clinical bedside.
• Adora2b Signaling on Bone Marrow Derived Cells Dampens Myocardial Ischemia-Reperfusion Injury.
• Adora2b-elicited Per2 stabilization promotes a HIF-dependent metabolic switch crucial for myocardial adaptation to ischemia.
• Ischemia and reperfusion--from mechanism to translation.
• The hypoxia-inflammation link and potential drug targets.
• Pressure controlled ventilation to induce acute lung injury in mice.
• Interplay of hypoxia and A2B adenosine receptors in tissue protection.
• Use of a hanging weight system for coronary artery occlusion in mice.
• [Unclear neurological condition and fever].
• Toll-like receptor signaling during myocardial ischemia.
• Signaling through the A2B adenosine receptor dampens endotoxin-induced acute lung injury.
• Role of extracellular adenosine in acute lung injury.
• Selective deletion of the A1 adenosine receptor abolishes heart-rate slowing effects of intravascular adenosine in vivo.
• PHD2 mutation and congenital erythrocytosis with paraganglioma.
• Central role of Sp1-regulated CD39 in hypoxia/ischemia protection.
• A2B adenosine receptor signaling attenuates acute lung injury by enhancing alveolar fluid clearance in mice.
• Usefulness of pressure-controlled ventilation at high inspiratory pressures to induce acute lung injury in mice.
• Hypoxia-inducible factor-1 is central to cardioprotection: a new paradigm for ischemic preconditioning.
• The reno-vascular A2B adenosine receptor protects the kidney from ischemia.
• Direct treatment of mouse or human blood with soluble 5'-nucleotidase inhibits platelet aggregation.
• The HIF2A gene in familial erythrocytosis.
• Physiological roles for ecto-5''-nucleotidase (CD73).
• Impact of ischemia and reperfusion times on myocardial infarct size in mice in vivo.
• A2B adenosine receptor dampens hypoxia-induced vascular leak.
• CD39/ectonucleoside triphosphate diphosphohydrolase 1 provides myocardial protection during cardiac ischemia/reperfusion injury.
• Lack of effect of extracellular adenosine generation and signaling on renal erythropoietin secretion during hypoxia.
• Identification of ectonucleotidases CD39 and CD73 in innate protection during acute lung injury.
• Contribution of E-NTPDase1 (CD39) to renal protection from ischemia-reperfusion injury.
• Cardioprotection by ecto-5'-nucleotidase (CD73) and A2B adenosine receptors.
• Upregulation of the water channel aquaporin-4 as a potential cause of postischemic cell swelling in a murine model of myocardial infarction.
• Protective role of ecto-5'-nucleotidase (CD73) in renal ischemia.
• ATP release from activated neutrophils occurs via connexin 43 and modulates adenosine-dependent endothelial cell function.
• Use of a hanging-weight system for isolated renal artery occlusion during ischemic preconditioning in mice.
• Nucleotide metabolism and cell-cell interactions.
• Systematic evaluation of a novel model for cardiac ischemic preconditioning in mice.
• Intraoperative metamizol as cause for acute anaphylactic collapse.
• Management of chronic obstructive pulmonary disease.
• The influence of mixed HCMV UL97 wildtype and mutant strains on ganciclovir susceptibility in a cell associated plaque reduction assay.
• Ganciclovir-resistant cytomegalovirus disease after allogeneic stem cell transplantation: pitfalls of phenotypic diagnosis by in vitro selection of an UL97 mutant strain.
• High impact of an expanded restriction fragment length polymorphism assay on detection of ganciclovir-resistant UL97 mutants of human cytomegalovirus.
• Rapid development of ganciclovir-resistant cytomegalovirus infection in children after allogeneic stem cell transplantation in the early phase of immune cell recovery.
• Drug-resistant human cytomegalovirus infection in children after allogeneic stem cell transplantation may have different clinical outcomes.
• Coordinate induction by antioxidants of UDP-glucuronosyltransferase UGT1A6 and the apical conjugate export pump MRP2 (multidrug resistance protein 2) in Caco-2 cells.

Schools

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• Eberhard Karls University TubingenGermany

Doctors Specialties

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• Anesthesiology

Accepted Insurances

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Awards

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• CMS Meaningful Use Stage 1 Certification

Education

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• Eberhard Karls University Faculty of Medicine

Hospital

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• University of Colorado Hospital

Helpful Reviews

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Medicare Facts

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National Provider Identifier [NPI]	1205012762
Last Name Of The Provider	ECKLE
First Name Of The Provider	TOBIAS
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