Professor of Medicine, Boston University
Cardiology
Dr. Wilson S. Colucci, MD
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Dr. Wilson S. Colucci works in the field of Cardiology. He studied medicine at Boston University School of Medicine. Boston University School of Medicine has a rank of 30 in research and a 52 in primary care He over the years received 4 awards: "Boston Magazine", "Elected Member", "Super Doctor" and "Fellow (FACC)". Wilson S. Colucci, MD has written publications. He has 66 publications published with PubMed. The lastest publication is: 'Impaired right ventricular hemodynamics indicate preclinical pulmonary hypertension in patients with metabolic syndrome.' He accepts Medicare.
Publications
- Lipopolysaccharide and cytokines inhibit rat cardiomyocyte contractility in vitro.
- High fat, high sucrose diet causes cardiac mitochondrial dysfunction due in part to oxidative post-translational modification of mitochondrial complex II.
- The lipid peroxidation product 4-hydroxy-trans-2-nonenal causes protein synthesis in cardiac myocytes via activated mTORC1-p70S6K-RPS6 signaling.
- Preclinical left ventricular diastolic dysfunction in metabolic Syndrome.
- Dysregulation of Intracellular Calcium Transporters in Animal Models of Sepsis Induced Cardiomyopathy.
- The Use of Digoxin in Patients with Worsening Chronic Heart Failure Reconsidering an Old Drug to Reduce Hospital Admissions.
- Does reversible cysteine oxidation link the Western diet to cardiac dysfunction?
- SERCA cysteine-674 sulphonylation and inhibition of L-type calcium influx contribute to cardiac dysfunction in endotoxemic mice, independently of cGMP synthesis.
- Relationship of plasma galectin-3 to renal function in patients with Heart failure: effects of clinical status, pathophysiology of Heart failure, and presence or absen...
- Cardiovascular Redox and Ox Stress Proteomics.
- Dynamics in insulin resistance and plasma levels of adipokines in patients with Acute decompensated and chronic stable heart failure.
- Mitochondrial transporter ATP binding cassette mitochondrial erythroid is a novel gene required for cardiac recovery after ischemia/reperfusion.
- Mitofusin-2 maintains mitochondrial structure and contributes to stress-induced permeability transition in cardiac myocytes.
- Relations of biomarkers of extracellular matrix remodeling to incident cardiovascular events and mortality.
- Oxidative posttranslational modifications mediate decreased SERCA activity and myocyte dysfunction in Galphaq-overexpressing mice.
- AFos inhibits phenylephrine-mediated contractile dysfunction by altering phospholamban phosphorylation.
- Redox-mediated reciprocal regulation of SERCA and Na+-Ca2+ exchanger contributes to sarcoplasmic reticulum Ca2+ depletion in cardiac myocytes.
- Cardiac-specific overexpression of catalase identifies hydrogen peroxide-dependent and -independent phases of myocardial remodeling and prevents the progression to ove...
- Episodes of Acute heart failure syndrome are associated with increased levels of troponin and extracellular matrix markers.
- Nesiritide enhances myocardial protection during the revascularization of Acutely ischemic myocardium.
- Nitroxyl activates SERCA in cardiac myocytes via glutathiolation of cysteine 674.
- Relations of matrix remodeling biomarkers to blood pressure progression and incidence of Hypertension in the community.
- Enhanced exercise capacity in mice with severe Heart failure treated with an allosteric effector of hemoglobin, myo-inositol trispyrophosphate.
- L-arginine fails to prevent ventricular remodeling and Heart failure in the spontaneously hypertensive rat.
- Parasympathetic nervous system and Heart failure: pathophysiology and potential implications for therapy.
- Effects of carvedilol early after myocardial infarction: analysis of the first 30 days in Carvedilol Post-Infarct Survival Control in Left Ventricular Dysfunction (CAP...
- Clinical and echocardiographic correlates of plasma procollagen type III amino-terminal peptide levels in the community.
- Direct myocardial effects of levosimendan in humans with left ventricular dysfunction: alteration of force-frequency and relaxation-frequency relationships.
- Role of reversible, thioredoxin-sensitive oxidative protein modifications in cardiac myocytes.
- Contemporary use of digoxin in the management of cardiovascular disorders.
- Vascular endothelial growth factor blockade promotes the transition from compensatory cardiac hypertrophy to Failure in response to pressure overload.
- The relationship between aldosterone, oxidative stress, and inflammation in Chronic, stable human heart failure.
- Cardioprotection: a new paradigm in the management of Acute heart failure syndromes.
- Aldosterone stimulates matrix metalloproteinases and reactive oxygen species in adult rat ventricular cardiomyocytes.
- Disruption of coordinated cardiac hypertrophy and angiogenesis contributes to the transition to Heart failure.
- Increased reactive oxygen species production and functional alterations in antioxidant enzymes in human failing myocardium.
- CD31- but Not CD31+ cardiac side population cells exhibit functional cardiomyogenic differentiation.
- A new hypertrophic mechanism of serotonin in cardiac myocytes: receptor-independent ROS generation.
- Alpha-adrenergic receptor-stimulated hypertrophy in adult rat ventricular myocytes is mediated via thioredoxin-1-sensitive oxidative modification of thiols on Ras.
- Mineralocorticoid receptor inhibition ameliorates the transition to myocardial failure and decreases oxidative stress and inflammation in mice with Chronic pressure ov...
- Biphasic temporal pattern in exercise capacity after myocardial infarction in the rat: relationship to left ventricular remodeling.
- Adiponectin-mediated modulation of hypertrophic signals in the Heart.
- Statins inhibit beta-adrenergic receptor-stimulated apoptosis in adult rat ventricular myocytes via a Rac1-dependent mechanism.
- Digoxin in the management of cardiovascular disorders.
- Relations of plasma matrix metalloproteinase-9 to clinical cardiovascular risk factors and echocardiographic left ventricular measures: the Framingham Heart Study.
- Landmark study: the Carvedilol Post-Infarct Survival Control in Left Ventricular Dysfunction Study (CAPRICORN).
- Relations of serum aldosterone to cardiac structure: gender-related differences in the Framingham Heart Study.
- Targeted anticytokine therapy in patients with Chronic heart failure: results of the Randomized Etanercept Worldwide Evaluation (RENEWAL).
- Pressure overload-induced myocardial hypertrophy in mice does not require gp91phox.
- Mice lacking osteopontin exhibit increased left ventricular dilation and reduced fibrosis after aldosterone infusion.
- Definition, classification, and staging of the adult cardiomyopathies: a proposal for revision.
- Myocyte contractile activity modulates norepinephrine cytotoxicity and survival effects of neuregulin-1beta.
- Regulation of matrix metalloproteinases by cytokines and reactive oxygen/nitrogen species in the myocardium.
- Hemodynamic and clinical effects of tezosentan, an intravenous dual endothelin receptor antagonist, in patients hospitalized for Acute decompensated heart failure.
- beta1 integrins expression in adult rat ventricular myocytes and its role in the regulation of beta-adrenergic receptor-stimulated apoptosis.
- Beta-blockers in Chronic heart failure.
- Beta-adrenergic receptor-stimulated apoptosis in cardiac myocytes is mediated by reactive oxygen species/c-Jun NH2-terminal kinase-dependent activation of the mitochon...
- Sustained hemodynamic effects of intravenous levosimendan.
- Endothelin mediates increased Pulmonary vascular tone in patients with heart failure: demonstration by direct intraPulmonary infusion of sitaxsentan.
- Exhaled nitric oxide: a marker of Pulmonary hemodynamics in heart failure.
- Molecular and cellular mechanisms of myocardial remodeling.
- Reciprocal modulation of mitogen-activated protein kinases and mitogen-activated protein kinase phosphatase 1 and 2 in failing human myocardium.
- Role of reactive oxygen species and NAD(P)H oxidase in alpha(1)-adrenoceptor signaling in adult rat cardiac myocytes.
- Nuggets, pearls, and vignettes of master Heart failure clinicians. Part 4--treatment.
- Impaired right ventricular hemodynamics indicate preclinical pulmonary hypertension in patients with metabolic Syndrome.
Schools
Boston University School Of Medicine
Peter Bent Brigham HospHarvard Med School
Peter Bent HospHarvard Med School
Procedures Preformed
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardioverter-Defibrillator or Pacemaker Insertion, Removal or Repair
- Chest CT (incl. Heart and Lungs)
Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Dissection
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
- View All
Doctors Specialties
- Cardiology
Accepted Insurances
Awards
- Boston Magazine
- Elected Member
- Super Doctor
- Fellow (FACC)
Education
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Brigham and Women's Hospital -
Boston University School of Medicine
Hospital
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Boston Medical Center
Drug Facts
| NPI NUMBER | 1588629505 | |
| NPPES Provider LastName | COLUCCI | |
| NPPES Provider FirstName | WILSON | |
| NPPES Provider ZIPCode | 021182309 | |
| NPPES Provider State | MA | |
| Specialty Description | Cardiology | |
| Total Claim Count | 96.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1588629505 |
| Last Name Of The Provider | COLUCCI |
| First Name Of The Provider | WILSON |
| View All |
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