Epileptologist at Cleveland Epilepsy Center
Neurology
Dr. Zhong Ying, MD
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Dr. Zhong Ying practices Neurology. He researched medicine at Wayne State University School of Medicine. Wayne State University School of Medicine is ranked 63 in research and 85 in primary care medicine He received awards:"CMS Meaningful Use Stage 1 Certification", "TopScholar Epilepsy Fellow Travel Grant" and "American Heart Association Fellowship Award". Zhong Ying is a published doctor and has 36 publications published. The lastest was: 'Glutamate receptor mechanisms in human epileptic dysplastic cortex.' He is registered with Medicare.
Publications
- Underlying Cortical Dysplasia as Risk Factor for Traumatic Epilepsy: An Animal Study.
- Growth-associated protein 43 and progressive Epilepsy in cortical dysplasia.
- Glutamate clearance mechanisms in resected cortical dysplasia.
- Neuronal nitric oxide synthase expression in resected epileptic dysplastic neocortex.
- Dissociation between in vitro and in vivo epileptogenicity in a rat model of cortical dysplasia.
- Expression of neural stem cell surface marker CD133 in balloon cells of human focal cortical dysplasia.
- The NMDA receptor NR2B subunit contributes to epileptogenesis in human cortical dysplasia.
- Temporal lobe volumes in patients with hippocampal Sclerosis with or without cortical dysplasia.
- Severity of histopathologic abnormalities and in vivo epileptogenicity in the in utero radiation model of rats is dose dependent.
- Mechanisms of epileptogenicity in cortical dysplasias.
- Increased numbers of coassembled PSD-95 to NMDA-receptor subunits NR2B and NR1 in human epileptic cortical dysplasia.
- Gene expression profile of neurodegeneration induced by alpha1B-adrenergic receptor overactivity: NMDA/GABAA dysregulation and apoptosis.
- Neocortical temporal FDG-PET hypometabolism correlates with temporal lobe atrophy in hippocampal Sclerosis associated with microscopic cortical dysplasia.
- Epileptogenicity of focal malformations due to abnormal cortical development: direct electrocorticographic-histopathologic correlations.
- Interictal EEG, hippocampal atrophy, and cell densities in hippocampal Sclerosis and hippocampal Sclerosis associated with microscopic cortical dysplasia.
- Focal cortical dysplasias in eloquent cortex: functional characteristics and correlation with MRI and histopathologic changes.
- Mechanisms of epileptogenicity in focal malformations caused by abnormal cortical development.
- Fluid-attenuated inversion recovery: correlations of hippocampal cell densities with signal abnormalities.
- Temporal lobe Epilepsy due to hippocampal sclerosis in pediatric candidates for Epilepsy surgery.
- Mechanisms of epileptogenesis.
- Brain plasticity and cellular mechanisms of epileptogenesis in human and experimental cortical dysplasia.
- Epileptogenicity correlated with increased N-methyl-D-aspartate receptor subunit NR2A/B in human focal cortical dysplasia.
- NMDAR1 receptor proteins and mossy fibers in the fascia dentata during rat kainate hippocampal epileptogenesis.
- Focal cortical dysplasia in children.
- Hippocampal chemical anatomy in pediatric and adolescent patients with hippocampal or extrahippocampal Epilepsy.
- NMDA-receptors 1 and 2A/B coassembly increased in human epileptic focal cortical dysplasia.
- Selective coexpression of NMDAR2A/B and NMDAR1 subunit proteins in dysplastic neurons of human epileptic cortex.
- Decreased calmodulin-NR1 co-assembly as a mechanism for focal Epilepsy in cortical dysplasia.
- Glutamate receptor mechanisms in human epileptic dysplastic cortex.
- Increased densities of AMPA GluR1 subunit proteins and presynaptic mossy fiber sprouting in the fascia dentata of human hippocampal Epilepsy.
- Induced expression of NMDAR2 proteins and differential expression of NMDAR1 splice variants in dysplastic neurons of human epileptic neocortex.
- AP-1 DNA binding activity induced by hyperosmolality in the rat hypothalamic supraoptic and paraventricular nuclei.
- Safe surgery of lesions near the motor cortex using intra-operative mapping techniques: a report on 50 patients.
- Motor and somatosensory evoked potentials in coma: analysis and relation to clinical status and outcome.
- [Mapping in tumor surgery of the central region].
Procedures Preformed
- EEG (Electroencephalogram)
- EMG (Electromyography)
Conditions Treated
- Alzheimer's Disease
- Ataxia
- Brain Aneurysm
- Cerebral Artery Thrombosis
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Doctors Specialties
- Clinical Neurophysiology
- Neurology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- TopScholar Epilepsy Fellow Travel Grant
- American Heart Association Fellowship Award
Education
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Cleveland Clinic Foundation -
Wayne State University School of Medicine -
Capital University of Medical Sciences
Hospital
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Cleveland Clinic
Drug Facts
| NPI NUMBER | 1871768697 | |
| NPPES Provider LastName | YING | |
| NPPES Provider FirstName | ZHONG | |
| NPPES Provider ZIPCode | 44195 | |
| NPPES Provider State | OH | |
| Specialty Description | Neurology | |
| Total Claim Count | 204.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1871768697 |
| Last Name Of The Provider | YING |
| First Name Of The Provider | ZHONG |
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